Which pharmacological action does Moxifloxacin predominantly feature?

Moxifloxacin is a type of fluoroquinolone antibiotic that primarily works by inhibiting two critical enzymes, DNA gyrase and topoisomerase IV. These enzymes play essential roles in bacterial DNA replication and maintenance. Inhibition of DNA gyrase prevents the supercoiling necessary for DNA replication, while inhibition of topoisomerase IV interferes with the separation of replicated DNA strands, which is crucial during cell division. Through this dual inhibition mechanism, Moxifloxacin effectively halts bacterial growth and replication, making it a potent agent against a variety of bacterial infections.

Other options represent different mechanisms of action characteristic of other drug classes. For instance, inhibiting cell wall synthesis is typical of beta-lactam antibiotics like penicillin. Meanwhile, inhibiting protein synthesis is a feature of antibiotics such as macrolides and tetracyclines. Altering membrane permeability is a general action seen in certain antibiotics but not specifically attributed to Moxifloxacin. Therefore, the predominant pharmacological action of Moxifloxacin is accurately described by its inhibition of DNA gyrase and topoisomerase IV.